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Demo version — illustrative data for a 52-year-old male. All values are representative examples. Built with the Health Tracker app.
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High SHBG Is Suppressing Free Testosterone
SHBG at 89–95 nmol/L (optimal 20–50) is binding most testosterone, leaving insufficient free T despite a strong total T of 902 ng/dL. Free T has declined from 35.6 pg/mL (Oct '24) to 26.2 pg/mL (Feb '26). Low zinc and sub-optimal DHEA-S are likely contributors. This is the most actionable hormone finding.
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LDL Borderline Elevated — 143–152 mg/dL
LDL has been above optimal (<100 mg/dL) across both test years. ApoB at 88 mg/dL is approaching the upper boundary — the most clinically meaningful cardiovascular risk marker. Lp(a) is reassuringly low at 13.4 mg/dL.
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Homocysteine at 11.8 µmol/L — Above Optimal
Within the standard lab range (<30) but above the optimal functional target (<10 µmol/L). Elevated homocysteine is associated with endothelial damage and cardiovascular risk. Typically responsive to methylated B12, methylfolate, and B6.
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Zinc Below Lab Minimum — May Be Affecting SHBG & Testosterone
Serum zinc at 66.2 µg/dL (lab minimum 70). Zinc is a direct cofactor in testosterone synthesis and known to inhibit aromatase. Adequate zinc is also required for normal SHBG regulation. An actionable and low-cost intervention.
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Inflammation Markers — Excellent
hs-CRP at 0.41 mg/L (low cardiovascular risk) and ESR at 7 mm/hr. Both in the optimal range. Low systemic inflammation is a highly protective long-term health signal.
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Blood Sugar — Optimal and Improving
HbA1c improved from 5.4% → 5.0% over four months. Fasting glucose at 82 mg/dL. Excellent insulin sensitivity with no diabetes or metabolic syndrome risk.
Key Metrics — Latest (Feb 2026)
🔍 Key Story — SHBG Is the Bottleneck
Total testosterone is excellent at 902 ng/dL — top quartile for most men. The problem is chronically elevated SHBG (89–95 nmol/L), which tightly binds testosterone and makes it unavailable to tissues. Free testosterone — the biologically active fraction — has declined from 35.6 pg/mL (Oct '24) to 26.2 pg/mL (Feb '26) despite total T rising. Factors that raise SHBG include low zinc, oestrogen excess, thyroid variation, and genetic expression. DHEA-S at 3.64 µmol/L is also below optimal, which can reduce androgen bioavailability.
Unit note: Free T is reported in pg/mL equivalents across both labs (reference range 9.57–40.6 confirms equivalence). Values are directly comparable.
🔍 Inflammation Overview
Direct inflammatory markers (hs-CRP, ESR) are excellent — low systemic inflammation is a highly protective finding. The area to address is homocysteine at 11.8 µmol/L. Standard lab ranges go up to 30, so this often goes unremarked — but the optimal functional target is <10. Homocysteine damages endothelial cells directly and is associated with increased cardiovascular and cognitive risk. Primary intervention: methylcobalamin (active B12), methylfolate, and P5P (active B6).
🔍 Lipid Profile Assessment
Positives: HDL is strong (68–74 mg/dL), triglycerides are healthy (58–71 mg/dL), and Lp(a) is low (13.4 mg/dL — this genetic risk factor is not a concern). TC/HDL ratio is reasonable.
Watch: LDL at 143–152 mg/dL is above optimal (<100 mg/dL) across both test years. ApoB at 88 mg/dL approaches the upper boundary. ApoB directly counts atherogenic lipoprotein particles and is now considered the superior cardiovascular risk marker to LDL-C alone. Homocysteine compounds this risk by damaging arterial walls.
Context: Given excellent triglycerides and high HDL, LDL particles may be predominantly large pattern-A (less atherogenic). The ApoB value helps contextualise this — at 88 mg/dL it is more reassuring than a raw LDL figure would suggest.
🔍 Metabolic Health — Outstanding
HbA1c improved from 5.4% → 5.0% — now firmly in the optimal range (<5.4%). Fasting glucose at 82 mg/dL is excellent. This confirms no insulin resistance or metabolic syndrome risk, and suggests elevated LDL is dietary or genetic in origin rather than insulin-resistance driven.
🔍 Thyroid Assessment
Thyroid function is healthy throughout. TSH moved from 1.12 (Oct '25) to 1.48 µIU/mL (Feb '26) — both within the optimal functional range (1.0–2.5). Free T3 and T4 (Oct '25) were comfortably within reference range. Thyroid status can influence SHBG levels — worth monitoring in parallel with hormone tracking.
🔍 Liver Health — Good
All liver enzymes within healthy ranges. AST (22), ALT (21), and GGT (14) are optimal. ALP was slightly low in Oct '25 (41 IU/L vs reference >43) but normalised to 67 in Feb '26. Low ALP can be an early signal of zinc or magnesium insufficiency — both of which are slightly suboptimal in this panel. The normalisation is a positive trend.
🔍 Kidney Assessment
Good kidney function throughout. Creatinine improved from 1.05 → 0.99 mg/dL. eGFR at 94 is comfortably above the ≥90 optimal threshold. BUN normalised from 18 (Oct '25) to 16.5 mg/dL (Feb '26). Uric acid is well controlled at 5.1 mg/dL — no gout risk currently.
🔍 Nutrient Status
Zinc (66.2 µg/dL): Below lab minimum of 70. Directly linked to testosterone synthesis and SHBG regulation — an actionable deficiency.
Vitamin D: Dropped from 92.4 (Oct '25 — above optimal upper bound of 80) to 54.3 ng/mL (Feb '26 — now well within optimal 40–80). Good correction, current dose appears appropriate.
Ferritin (167 ng/mL): Within range (22–322) but in the upper half — worth monitoring at the next test. Ferritin is also an acute-phase reactant.
Magnesium (2.21 mg/dL): Within optimal range and trending up — good. Magnesium supports testosterone production, sleep, and muscle recovery.
🔍 Blood Count Pattern
There is a consistent pattern across both test years: neutrophils slightly below reference (~39%, ref ≥40%) and lymphocytes slightly elevated (~44%, ref ≤40%). This pattern can be a benign constitutional variant but may also reflect chronic immune activation. Absolute counts are within range. Haemoglobin at 13.9–14.2 g/dL is within normal range for men, though tracking towards the lower boundary — worth monitoring alongside zinc and iron levels.